Monograph 2
Fundamentals of Initial Case
Management Following Trauma
By R. C. Schafer, DC, PhD, FICC
Manuscript Prepublication Copyright 1997
Copied with permission from
ACAPress
Without a doubt, no other health-care
approach equals the efficacy of chiropractic in the general field
of conservative neuromusculoskeletal rehabilitation.
For many centuries, therapeutic rehabilitation was a product
of personal experience passed on from clinician to clinician. In
the last 20 years, however, it has become an applied science. In
its application, of course, much empiricism remains that can be
called an intuitive art --and this is true for all forms of
professional health care.
INTRODUCTION
The word trauma means more than the injuries so common with falls, accidents, and
contact sports. Taber1 defines it as "A physical
injury or wound often caused by an external force or violence" or
"an emotional or psychologic shock that may produce disordered
feelings or behavior." This is an extremely narrow definition for
trauma can also be caused by intrinsic forces as seen in common
strain. In addition to its cause being extrinsic or intrinsic,
with a physical and emotional aspect, it also can be the result
of either a strong overt force or repetitive microforces. This
latter factor, so important in treating a unique patient's
specific pathophysiology, is too often neglected outside the
chiropractic profession.
Taber1 states rehabilitation is "The process of
treatment and education that lead the disabled individual to
attainment of maximum function, a sense of well being, and a
personally satisfying level of independence. The person requiring
rehabilitation may be disabled from a birth defect or from an
illness. The combined effects of the individual, family, friends,
medical, nursing, allied health personnel, and community
resources make rehabilitation possible." It is surprising that
Taber excludes trauma as a prerequisite for rehabilitation for it
is the most common factor involved.
Other authors define rehabilitation strictly in terms of
exercise and restorative therapeutic modalities and regimens.
Some limit the term to preventing or reversing the noxious
effects of the inactivity or lessened activity associated with
the healing process. While it is true that these definitions hold
significant components of clinical reconditioning and
restoration, the scope of rehabilitation means much more to the
chiropractic physician.
It has been the custom of the majority health-care profession
not to consider rehabilitative procedures until late in case
management. Like Welch2, allopaths generally place it
6th in the cycle of managing the body's response to injury:
(1) injury
(2) pain, bleeding and traumatized tissue
(3) inflammation
(4) repair and regeneration
(5)
atrophy
(6) rehabilitation.
This author asks, "Why wait
for signs of atrophy to begin rehabilitation? Nonparalytic
atrophy beacons a neglected patient."
Throughout many of my manuscripts, emphasis is placed on
minimizing the noxious effects of fibrosis. Fibrosis parallels
atrophy and leads to
(1) impaired cellular nutrition and drainage,
(2) stiff, shortened soft tissues,
(3) trigger-point development,
(4) adhesion development, and (5) articular fixations restricting normal ranges of motion.
Restricted joint mobility, in turn, encourages further
atrophy, stasis, and a lack of mechanoreceptor input. Thus, a
vicious cycle is established leading to a greater risk of
residual impairment, reinjury, and progressive degeneration.
After bleeding and pain are controlled, a primary objective is
the normalization of soft-tissue flexibility, elasticity, and
pliability as soon as possible.
Posttraumatic rehabilitative procedures ideally begin at the
time the doctor first sees the patient. Hopefully, this will be
an early stage --one occurring soon after injury. Alert care will
usually control the ill effects of inflammation, enhance repair
and regeneration mechanisms, and halt, if not nullify, the
progress of atrophy and the formation of fibrosis. In many cases,
customary surgery may be avoided. With individualized care, the
result is a greater likelihood of obtaining an optimal goal of
full function, strength, power, resistance, agility, and
endurance.
THE APPROACH
Experienced clinicians
recognize the need to treat the entire kinematic chain and
related functions involved in an injury and not a particular
joint or part manifesting acute symptoms. Protocols, templates,
and regimens are used throughout many manuscripts simply as
guidelines. They should not be considered "the standard" or "the
only way to do it." Every person is unique in many ways. The way
each responds to trauma and pain and its treatment is no
exception. Thus, a "cookbook" approach to rehabilitative therapy
is irrational if suggestions are taken as a directive rather than
a framework for thought --a framework, not a cage.
Most texts concerning rehabilitative therapy directed to
allied health-care professions appear to be developed from the
standpoint of a perfectly healthy individual who has suffered an
injury. The young practitioner should keep in mind that this is
rarely the case. The typical traumatized patient presents with an
array of underlying overt and subclinical pathologic,
dyfunctional, and emotional disorders that must be considered in
arriving at a practical treatment plan.
An examiner should keep in mind that trauma is not always the
cause of all an injured patient's complaints. A painful injury
may only be the precipitating factor bringing the patient to the
doctor's office. Trauma produces overstress, and overstress
frequently brings out subclinical disorders because of the tax on
the immunologic system and other body reserves. The
differentiation of the immediate from predisposing factors is
just one component of the clinical art.
Physician Responsibilities
If a doctor were to concern
himself solely with injury prevention, care, and rehabilitation,
his role would be much easier. But many factors are involved. For
instance, consider patient motivation and cooperation -without
which case management is an uphill battle.
The average patient bears many pressures. These pressures may
blind the patient to the fact that continuing usual activities at
this time may make a minor condition worse or that the
continuation of treatment after pain has subsided is necessary to
reach optimal goals.
Physical activity beyond the point of exhaustion or tissue
strength or continuing stressful activity with an injury where
further insult may lead to permanent injury is illogical from a
clinical viewpoint but common behavior. And for various reasons,
some people may avoid reporting injury or even try to hide its
effects.
The attending physician should mentally target that he is only
responsible to the patient and his professional code of conduct.
He is not responsible to any other person except the parent of a
minor or a legal guardian. Thus the questions must be asked: Who
has the authority to return an injured employee to work: the
attending physician, the company doctor, or the patient's
employer? Who has the authority to return an injured athlete to
play or practice: the attending physician, the trainer, or the
coach?
BASIC DIAGNOSTIC AND SUPPORT PROCEDURES
This and other monographs in
this series describe basic diagnostic procedures, offer a brief
review of fundamental disorders, and explain the basic clinical
template on which all subsequent papers are framed. The topic
begs an explanation of physiologic performance, conditioning
rationale, and exclusion criteria for potentially harmful
activity.
Early Case Management Plan
Each doctor usually has a
general case management plan that can be readily adapted to the
situation at hand. An example is listed below.
Survey the situation for a medical emergency, and take
whatever first-aid measures are necessary, record patient status
and vital signs, and record as much of a detailed history as
logical under the circumstances.
Record findings of inspection and tolerated palpation and
movement of the part involved. Auscultate involved areas for
crepitus; observe abnormal limb, spinal, pelvic, and rib
positions suggesting fracture or dislocation; and seek signs of
CNS injury or internal hemorrhage.
Record integrity of normal superficial and deep reflexes
and occurrence of pathologic reflexes, and order roentgenography
if fracture or dislocation is suspected.
Record findings of involved joint range of motion studies
and muscle strength to the degree possible at the stage existing
at the time of examination, order electromyographic and/or
thermographic studies as indicated, and order pertinent
laboratory studies.
Coordinate findings, patient's objectives, and prognosis,
and set clinical goals, treat and monitor, periodically reassess
patient progress with the working diagnosis and clinical goals,
and retest when confirmation is necessary.
Recording oral temperature, comparing extremity pulses and
limb blood pressure, and auscultating the heart and lungs should
be a common routine in any examination. Traumatic injury is no
exception. However, the state of the patient must set
priorities.
The History Interview
The personal data collected
should be similar to that recorded routinely such as name,
address, date of birth, age, sex, present complaints, present and
past occupations, type of work and the number of hours,
recreational activities, medical and surgical history, accidents
and injuries history, drug and food sensitivities, allergies,
congenital difficulties, diet, smoking and drinking habits,
insurance data, etc. The history should also define the types of
primary and secondary physical activities, the number of hours
involved, the level of achievement, the age at which active
involvement began, etc.
A thorough history forms the basis for differentiating acute
from chronic disorders. See Table 1.
Table 1. Differentiation of Acute and Chronic Disorders
Clinical Finding |
Acute Inflammation |
Chronic Inflammation |
Pain |
Relatively constant; likely referred
over a diffuse segmental area. In intrinsic disorders, pain is
increased on movement in any direction. In periarticular
disorders, pain is increased on movement only in certain
planes. |
Increased by specific movements,
relieved by rest; likely to be relatively localized near, but not
necessarily over, the site of the lesion. |
Passive motion |
Muscle spasm or empty end-feel at the
end of motion. |
No muscle spasm or empty end-feel at the
end of motion, possible blocking. |
Tenderness |
Severe. |
From slight to moderate. |
Skin temperature |
Measurable increase. |
No measurable increase. |
Sleeping pattern |
Difficulty in falling asleep, staying
asleep, or both. |
No sleeping difficulty unless a hip or
shoulder is involved. |
Typical Early Findings
Typical overt changes
(traumatic or nontraumatic) commonly discovered in disorders of
the musculoskeletal system include:
- Color changes such as ecchymosis and redness
- Local heat
- Swelling from synovial thickening, periarticular edema,
nodules, or bony enlargement
- Deformity from abnormal bone angulation or subluxation
- Wasting from atrophy or dystrophy
- Tenderness on palpation
- Pain on motion
- Limitation of motion
- Joint instability
- Carriage and gait abnormalities.
In examining a patient in pain, certain types of pain are
clinically significant. For example, a sharp severe pain
associated with muscle changes and sensory disturbances radiating
along the distribution of a nerve is characteristic of acute
nerve compression. Pain from fracture is severe, throbbing, and
acutely aggravated by movement of the part.
Spine-Board Transportation of the Injured
Moving an injured person
frequently requires extreme care. In moving a severely injured
person from one location to another, a spine board is more
appropriate than the common stretcher because the board helps to
prevent further aggravation of a possible vertebral or spinal
cord lesion. Craig 3 warns that transporting an injured
athlete from the field must be well-planned, orderly, and
conducted in an unhurried manner.
Four general steps are involved
in transporting any injured person:
The patient should be placed supine with care taken that
the head, neck, and spine are in normal alignment. The spine
board is placed close and parallel to the patient's body. Note,
however, that a conscious patient with a painful disorder is
understandably reluctant to change a position that seems
comfortable, thus tending to favor a position usually involving
minimum movement.
Usually, the patient's arm next to the board is placed at
the subject's side and the patient's other arm is extended over
the head if it is not injured. The patient is then gently rolled
onto the side opposite the patient's extended arm. The patient
should be rolled like a "log" to maintain body alignment. An
exception to this would be suspicion of a spinal fracture,
whereon the player should be kept on the side and not rolled
supine. This may require seven people in a spinal or head injury,
otherwise two people will usually suffice unless the player is
extremely large.
The spine board is inserted under the back of the patient
(not under the patient's side). Then, gently roll the patient
onto the spine board. In cases of unconsciousness, facial or
mouth fractures, bleeding from the mouth or nose, the patient is
kept laterally recumbent to allow drainage and an open
airway.
Assure that the injured person is in a comfortable
position, then snug straps around the board and patient to secure
the injured person to the spine board during transportation. If
possible, remove rings from injured hands before swelling
occurs.
Emergency Immobilization by Splints
To prevent further damage
during referral, a fractured bone should be immobilized by
immediately splinting the joints above and below the fracture
because movement of these joints would disturb the bony segments.
The splint should be well padded to protect the skin from injury,
loss of circulation, inflammation, and infection.
A pneumatic inflatable splint is especially useful in limb
fracture because it allows both immobilization and compression to
minimize effusion and hemorrhage. It must be applied only snug
enough to support any fracture fragments without inhibiting
circulation.
To immobilize a fractured bone in the thigh or hip, an
improvised splint must extend from the groin and the armpit to
several inches below the foot. Padding should extend over the
ends of the splint at the groin and the armpit. The bandages or
straps used to secure a splint must not be applied so tightly
that they impair circulation even for a few minutes. A bluish
discoloration of the nailbeds or skin of the affected limb would
suggest that one or more bandages are too taut. Security bandages
should never be tied directly across the site of injury.
A dislocation is immobilized during referral in the same way
as a fracture: close to the joint. Area ligaments are usually
torn and may require surgical repair. Cold compresses can be
applied to the area to relieve pain and reduce swelling. The
patient's temperature must not be lowered because hyperthermia
invites shock.
Postreduction immobilization of a dislocation in the lower
extremity usually requires 6 weeks and in the upper extremity
requires 3 weeks. Inadequate care, especially for ankle and
shoulder dislocations, leads to chronic weakness, movement
restrictions, instability, and recurrent dislocation in which
subsequent surgery has a poor prognosis in restoring preinjury
status. Except for recurring dislocations, almost all overt
dislocations require anesthesia before reduction.
Roentgenology
Roentgenography should be
used to confirm or dispose of suspicions arising during the
history and physical examination, and not used as the sole basis
of the diagnosis. When a film is used alone to confirm a prior
clinical opinion, other clues exhibited on a view may be missed
that indicate a different approach. This occurs when an
outstanding feature, visible at a distance, overwhelms a desire
to seek other evidence. Nevertheless, whatever is presented on
the film must be evaluated; eg, an asymptomatic chronic disease
process may be underlying an acute injury.
X-ray films are often helpful in determining dislocations,
overt fractures, stress fractures, joint-space alterations,
ossification, calcification, and sometimes cartilage fractures,
fat pad alterations, and masses and swelling. Common radiographic
signs of various bone lesions are shown in Table 2. Once relevant
features classify an abnormality, a search should be made for
details enabling it to be distinguished from others in the same
class. This takes careful evaluation of frequently subtle
soft-tissue changes which confirm osseous alterations.
Table 2. Radiologic Signs of Various Bone Lesions
Infection |
Singular Malignant Bone Lesion |
Formation of sequestration and
involucrum |
Permeactive or moth-eaten destruction
(wide transition zone) |
Irregular periosteal reaction frequent,
no speculation |
Extraosseous extension with soft-tissue
mass, occasional fluffy calcifications |
Diaphyseal site most
common |
Irregular (sometimes spiculated)
periosteal reaction |
Variable bone destruction
|
Metaphyseal area is the most common
site |
Destruction of adjacent cartilage
crossing joints (most malignancies lack this
trait) |
|
Metastatic Lesions
|
Benign Bone Lesions |
Moth-eaten destruction of cortex and
medulla |
Enlargement of an intact
cortex |
Pathologic fractures |
Homogeneous periosteal
reaction |
Diaphyseal site most
common |
Sclerotic margins (narrow transition
zone) |
Periosteal reaction
absent |
Multiple bone
involvement |
The examiner must be well acquainted with the nature of all
substances visible on a film. This is a medicolegal
responsibility. Healthy tissue features and common variances
should be recognized at a glance. Joint abnormalities show
significant alterations in structure, symmetry, continuity,
positional relations, length and breadth, cartilaginous joint
space, and density. Calcareous density is much greater than
muscle density, fat density is much less than muscle density, and
gas density is far less than that of fat density.
In addition to roentgenography of one or more distressed
joints, spinal and chest films are almost always included if the
possibility of referred pain or systemic symptoms is
involved.
Skin and Tendon Reflexes
Evaluation of pertinent
superficial and deep tendon reflexes should be checked as a
standard procedure. Upper-limb tendon and periosteal reflexes are
supplied essentially by C5–T1 segments of the cord; lower-limb
reflexes, essentially by the L2– S3 segments. A summary of normal
reflexes is shown in Table 3.
Table 3. Summary of Normal Reflexes
Superficial Reflex |
Afferent Nerve |
Center |
Efferent Nerve |
Anal |
Pudendal |
S3–S5 |
Pudendal |
Consensual |
Optic |
Midbrain |
Oculomotor |
Corneal |
Trigeminal |
Pons |
Facial |
Cremasteric |
Femoral |
L1 |
Genitofemoral |
Lower abdominal |
T10–T12 |
Cord level |
T10–T12 |
Nasal (sneeze) |
Trigeminal |
Brainstem,
upper
cord |
Cranial V, VII, IX, X and spinal
nerves of respiration |
Plantar |
Tibial |
S1–S2 |
Tibial |
Upper abdominal |
T7–T10 |
Cord level |
T7–T10 |
Uvular |
Glossopha- ryngeal
|
Medulla |
Vagus |
Tendon or Periosteal Reflex |
Afferent Nerve |
Center |
Efferent Nerve |
Achilles |
Tibial |
S1–S2 |
Tibial |
Biceps |
Musculocu- taneous |
C5–C6 |
Musculocutan- eous |
Jaw jerk |
Trigeminal |
Pons |
Trigeminal |
Patellar |
Femoral |
L2–L4 |
Femoral |
Radial |
Radial |
C6–C8 |
Radial |
Triceps
|
Radial
|
C6–C7
|
Radial
|
Visceral Reflex |
Afferent Nerve |
Center |
Efferent Nerve |
Accommodation |
Optic |
Occipital |
Oculomotor |
Bulbocavernous |
Pudendal |
S2–S4 |
Pudendal |
Carotid sinus |
Glossopha- ryngeal |
Medulla |
Vagus |
Ciliospinal |
Sensory nerve |
T1–T2 |
Cervical
sympa- thetics |
Light |
Optic |
Midbrain |
Oculomotor |
Oculocardiac |
Trigeminal |
Medulla |
Vagus |
PAIN: GENERAL CONSIDERATIONS
All joints contain
nociceptors in their ligaments, tendon insertions, periosteum,
fibrocartilages (sparsely), capsules, and vascular walls.
Authorities differ whether or not synovia contains nociceptors;
most believe that it does not.
An extremely "ticklish" person is one whose superficial
reflexes (skin and muscle) are very lively, thus a low pain and
temperature threshold can be anticipated.
Significance of Hyperalgesia
A painful tenderness
produced by external pressure frequently results from traumatic
lesions of sensitive subdermal tissue, trigger points, the
development of a toxic accumulations, or a deep-seated
inflammatory irritation. Pottenger pointed out that hyperalgesia
of soft tissues is not uncommon in the areas that have been the
seat of reflex sensory pain. For example, subcutaneous soreness
within the shoulder and upper arm muscles is often associated
with inflammatory diseases of the lungs. He also reported that
cutaneous hyperalgesia is a common finding in visceral disease.
Hyperalgesic skin frequently overlies an area of pleurisy, a
tubercular cavity, a peptic ulcer, or an inflamed ovary. Zones of
hyperalgesia (often associated with precapillary vasoconstriction
and hypermyotonia) are more often associated with acute and
subacute visceral disease than with chronic disorders.
Characteristics of Extremity Pain
The cause of limb pain may
be of mechanical, chemical, thermal, toxic, nutritional,
metabolic, or circulatory origin, or a combination of several of
these factors depending on the pathologic or traumatic process
involved. Peripheral nerve disease will sometimes reveal a
history of an entrapment neuropathy. Nutritional disorders can
result in a polyneuropathy because of unfavorable metabolic
activities within the neural apparatus.
When an inflammatory process involves sensory fibers, the pain
is frequently perceived along the total course of the nerve. The
pain may be referred along a somatic dermatome because of
visceral inflammation, ischemia, or a tumor (eg, the shoulder-arm
pain associated with myocardial infarction or angina). Such pains
have two major features in common:
(1) their distribution is limited to an anatomical dermatomal pattern and
(2) interruption of the nerve s function by any means alleviates the symptoms (at
least temporarily).
Characteristics of Neuralgia
Neuralgia is any sharp,
severe, stabbing, paroxysmal, remittent pain with temporary
abatement in severity that travels along the course of one or
more nerves. It is usually associated with tenderness along the
course of the nerve and violent episodic spasms in the muscles
innervated.
Pain accentuated by heat points to neuritis or congestion. In
contrast, pain that is relieved by heat suggests something
producing abnormal myotonia or possibly ischemia. Pain of
intrinsic neurologic origin is generally accompanied by
paresthesias and root signs. When throbbing pain occurs, vascular
engorgement, crush syndrome, a vasomotor disturbance, or possibly
Paget s disease should be the first suspicions. It s difficult
for the patient to describe its character because it is unlike
any other type of pain and usually is a combination of painful
sensations.
Neuralgia is provoked by any peripheral stimulation in the
involved zone, and stimulated trigger points cause spontaneous
paroxysms. Morphologic changes cannot be detected early in a pure
neuralgia or neurodynia syndrome. The term neurodynia
describes a similar pain that is less severe; ie, a deep ache.
The patient vigilantly guards the involved part and shows great
apprehension. The pain manifests in the involved nerve s
distribution superficially or deep. It usually radiates, and
there is an exorbitant response to stimulation.
Neuralgia rarely subsides spontaneously. It is often so severe
that the victim becomes totally incapacitated and frequently
addicted to narcotics. Depression is often associated, and
suicidal tendencies sometimes arise.
REFERENCES
1. Thomas CL (ed):
Taber's Cyclopedic Medical Dictionary, ed 14.
Philadelphia, F.A. Davis, 1981.
2. Welch B: The Injury Cycle. Sports Medicine Update,
1:1, 1986.
3. Craig TT (ed): Athletic Injuries and First Aid: Comments
in Sports Medicine. Chicago, American Medical Association,
1973.
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